AC-262536 activates androgen receptors in muscle cells, triggering protein synthesis and inhibiting muscle breakdown. Unlike testosterone, it avoids binding to sex hormone-binding globulin (SHBG), allowing more free molecules to remain active. This mechanism promotes nitrogen retention and accelerates muscle repair post-workout.
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Studies suggest AC-262536 may upregulate follistatin, a myostatin inhibitor, further enhancing muscle hypertrophy. This dual-action approach sets it apart from other SARMs.
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